This is a quick, clinic-facing overview of NICE NG136 for day-to-day primary care. It focuses on the decisions that most often matter in consultations: confirming the diagnosis, spotting severe hypertension, who to treat, how to start/step-up meds, and what targets to aim for.
Key clinician “anchor points” (remember these)
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Confirm before you label: clinic 140/90–179/119 → confirm with ABPM (preferred) or HBPM before diagnosing hypertension.
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Severe BP is a safety pathway: clinic ≥180/120 → assess for target-organ damage; urgent/same-day referral if damage is present or strongly suspected (don’t wait for ABPM/HBPM).
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Stage 1 meds = risk-led (but don’t ignore younger patients): if <80 with stage 1 plus target-organ damage / CVD / CKD / diabetes / 10-yr CVD risk ≥10% → offer treatment; if ↓risk <10%, still consider meds, especially if younger and persistent.
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Step-1 drug rule (A/C): <55 and not Black African/African-Caribbean → ACEi/ARB; ≥55 or Black African/African-Caribbean → CCB, then step up by adding ACEi/ARB or thiazide-like diuretic stepwise.
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Targets + resistant HTN mantra: aim clinic <140/90 (<80) or <150/90 (≥80) (with lower ABPM/HBPM targets); individualise in frailty and use standing BP if postural drop. If “resistant” on ACEi/ARB + CCB + thiazide-like → confirm with ABPM/HBPM + check adherence, then add spironolactone if K ≤4.5 (or α/β-blocker if not suitable), and seek specialist advice if still uncontrolled.
Read-with-care notes (details omitted for brevity in this quick guide)
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ABPM validity: ensure ≥14 daytime (waking) readings to count as a valid diagnostic ABPM set.
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Baseline testing: include dipstick haematuria as well as urine ACR.
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Eyes: offer fundoscopy to all patients with confirmed hypertension (not only when you feel it’s “clinically indicated”).
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First visit technique: measure both arms initially; if a persistent >15 mmHg difference exists, use the higher arm for ongoing readings.
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Postural hypotension definition: a significant postural drop is ↓SBP ≥20 mmHg or ↓DBP ≥10 mmHg after standing for ≥1 minute (the diastolic threshold is easy to miss but clinically important).
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Full severe hypertension referral criteria and detailed drug titration steps are covered in the accompanying detailed print charts.
Conclusion
Use NG136 as a reliable “workflow”: confirm out-of-office, treat severe readings as urgent, start meds when risk/organ damage justifies it, apply the A/C step-1 rule, and be systematic when BP looks resistant (confirm + adherence + contributors before escalating). This keeps decisions safe, consistent, and quick in real GP clinics.
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Reference
